How the body develops immunity
against an infection
A new antibody study suggests that
immunity to Covid-19 can be lost in months, but body notes are not the only way
the body develops immunizations (immunity). What is the alternative and does it limit the
study? or how to improve immunity.
The recommendation is based on a high drop in
patient resistance levels within three months after infection. Looking at the
findings, results and limitations of new research, and the broad question of
how the body grows and the vulnerability of infection or how to improve immunity:
What
are the findings of the study?
Researchers are analyzing the immune
response (immunity) of 90 diagnosed patients and Covid-19 patients and health workers. In
their study, which has not yet been peer-reviewed, they found that antibodies
directly contribute to the depletion of the coronavirus SARS-CoV-2 by 2-23 over
a period of 18-65 days. This is similar to the decrease in antibodies detected
in seasonal coronaviruses associated with the common cold.
They suggested that the immune
system (immunity) formed by the formation of antibodies against SARS-CoV-2 lasts only a
few months, and that Covid-19 patients who are diagnosed are more likely to be
diagnosed and more likely to become infected again.
New Research: Lessons from immune response system (immunity) of most severe Covid-19 patients
The
analysis found a “strong level” of antibodies produced in 60% of participants
at the time of their infection and only 16.7% maintain that level of potency
for 65 days over time. While the level of antibodies is still high in patients
with severe symptoms, researchers have said it is unclear why the antibody
response (immunity) is associated with the severity of the disease. Researchers have noted
that asymptomatic individuals also develop antibodies to fight the virus.
What
are the implications and the limitations of these findings?
Researchers say that this study has
significant implications for the more comprehensive serological tests, the more
likely it is that a person will become infected with SARS-CoV-2, and the
stronger the immune system (immunity).
Research
is needed to find out how antibodies are needed to protect against infection or how to improve immunity.
”“ We need to know more about the antibody titer needed to prevent re-infection
in humans.
Does a drop in antibodies result in losing immunity against the virus?
Not at
all. Finger-like antibodies give us evidence that the pathogen (coronavirus in
this case) has caused an infection and that the immune system (immunity) has responded.
Antibodies usually stay in the blood for a while and immediately activate the
immune system (immunity) when the body is exposed to the virus again. Some antibodies not
only detect when the pathogen returns, but also protect the body throughout
life from re-infection, as in the case of measles. However, in the case of
seasonal flu, antibodies offer very little protection.
Evidence of immunity is a glimmer, but data show lasting Covid-19 damage
In the case of the novel coronavirus, it
is not yet clear how long antibodies provide protection (immunity) against an infected
person. "More research is needed to determine the longevity of the
antibody response (immunity) and what level of antibodies are needed to prevent re-infection,"
the researchers said in a statement. Until then, the presence of antibodies
only gives us evidence that the person was exposed to the virus.
Researchers
warn that antibodies are one way the body can fight off a virus such as
SARS-CoV-2.
Drawing for covid-19 precautions
So,
what is the other way?
Investigators say, the role played by
T-cell responses through infection or vaccination game control”.
T cells are an important part of the
immune response (immunity). The human body has two mechanisms of non-invasive response
within the changing immune system (immunity). The first is the immune system (immunity), also called
immune deficiency and has been discussed above. However, if the virus enters
the cell and can no longer be detected by antibody-Mediated immune infections,
the immune response (immunity) can take a toll to kill the virus.
Cellular immunity occurs within the
infected cell, and it is targeted to cells called T lymphocytes. These are T
cells, which recognize the infected cell. Prior to that, these cells must work
in contact with the antigen (virus) - to introduce the cell. When T cells are
activated, they self-produce to form more T cells, killing the infected cells.
During activation, some T cells remain
inactive as memory cells. This produces more T cells when the infection
returns. Therefore, memory plays an important role in providing moderate physical
inactivity. There are also helper T cells, which work indirectly in contact
with other body cells about possible viruses.
What is the evidence so far in the immune
response (immunity) to SARS-CoV-2?
Data from
the immune response show that immunity between cells is also effective in some
cases and protects against T cells in the blood.
The doubts over herd immunity
Researchers from Germany found that
T cells accumulated over time in patients' infections. It said T cells-SV-CoV-2
cells were identified as "people exposed to other common colds",
which is why he suggested that the T-cell response found in healthy people may
be suppressed by T cells based on previous exposure.
U.S. investigators say T-cell reaction
against SARS-CoV-2 has been detected in non-revealing individuals.
"However, the source and clinical correlation of regeneration is still
known. It is thought that this reflects T cell memory in circulating the
'common flu' coronavirus.
U.S. researchers reviewed Covid-19
immunology also asked two questions about the T-cell response: first, what is
the contribution of T cells to initial viral control and tissue damage in the
Covid-19 source; secondly, T-cells that are developed later donate to the
immune system (immunity). The researchers wrote: "While the discovery of immune
defenses is not essential for effective control of the virus, dysfunctional T
cell responses can cause immunopathology and contribute to Covid-19
degeneration."